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Supporting this, our experimental work showed that the treatment of insulin-1 (INS-1) cells with high glucose-induced glucotoxicity, characterized by insulin suppression, reduced PDX-1 mRNA expression, loss of GSIS, increased ROS levels, and significantly elevated CD36 expression

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and in the remodeling phase, transforming growth factor TGF-β signaling coordinates collagen fiber deposition while suppressing excessive α-smooth muscle actin (α-SMA) expression, enabling scar-free healing within 14 days